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Individuals with increased inflammatory response to ozone demonstrate muted signaling of immune cell trafficking pathways

Creator: Fry, Rebecca, Rager, Julia E, Zhou, Haibo, Zou, Baiming, Brickey, June W, Ting, Jenny P.-Y., Lay, John C, Peden, David, Alexis, Neil

Abstract Background Exposure to ozone activates innate immune function and causes neutrophilic (PMN) airway inflammation that in some individuals is robustly elevated. The interplay between immuno-inflammatory function and genomic signaling in those (more)

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12931_2012_article_1339.pdf

Collection: BioMed Central

Date Deposited: 2016-06-03

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Additional File 1: Genes significantly differentially expressed after ozone challenge.

Collection: BioMed Central

Date Deposited: 2016-06-03

Date Created: 2016-06-03

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Figure 1 Ozone response for neutrophils differs between responders and non-responders

Collection: BioMed Central

Date Deposited: 2016-06-03

Date Created: 2016-06-03

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Figure 4 Summary of the ozone-induced changes in non-responders and responders

Collection: BioMed Central

Date Deposited: 2016-06-03

Date Created: 2016-06-03

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